Echauffement et blessures

Le sport pour bien vieillir

17/12/2011

 

L’athlète master d’endurance, un modèle de vieillissement réussi
Science & Sports 2012 Louis J

Objectifs
Cet article vise à présenter l’athlète master d’endurance à travers ses caractéristiques physiologiques et passe en revue l’effet de l’âge sur les principaux facteurs de la performance en endurance.

Actualités
Le déclin de la performance physique est inévitable avec l’avancée dans l’âge. Néanmoins, certaines personnes âgées continuent à s’entraîner autant que leurs homologues plus jeunes afin de repousser les effets du vieillissement. Ces « athlètes masters » présentent très peu d’altérations physiques, sont capables de performances remarquables dans les épreuves d’endurance et constituent ainsi de véritables modèles de vieillissement réussi. L’activité physique régulière leur permet de limiter l’altération des capacités physiologiques nécessaires à une bonne qualité de vie.

Perspectives
L’athlète master constitue un modèle expérimental intéressant dans l’étude du vieillissement, sans les effets confondus d’un mode de vie sédentaire et du déconditionnement qui accentuent le processus de vieillissement. Les recherches futures devraient ainsi inclure les athlètes masters afin d’enrichir les connaissances sur le processus de vieillissement et sur les stratégies bénéfiques pour bien vieillir.

S’entraîner trop léger ne renforce pas les tendons

28/11/2011

 

Après, tu passes à la muscu, et tu te fais mal partout

Expression of extracellular matrix components and related growth factors in human tendon and muscle after acute exercise
K. M. Heinemeier, S. S. Bjerrum, P. Schjerling, M. Kjaer
Scandinavian Journal of Medicine & Science in Sports Early View

Acute kicking exercise induces collagen synthesis in both tendon and muscle in humans, but it is not known if this relates to increased collagen transcription and if other matrix genes are regulated. Young men performed 1 h of one-leg kicking at 67% of max workload. Biopsies were taken from the patellar tendon and vastus lateralis muscle of each leg at 2 (n = 10), 6 (n = 11), or 26 h (n = 10) after exercise. Levels of messenger ribonucleic acid mRNA for collagens, noncollagenous matrix proteins, and growth factors were measured with real-time reverse transcription polymerase chain reaction. In tendon, gene expression was unchanged except for a decrease in insulin-like growth factor-IEa (IGF-IEa; P < 0.05). In muscle, collagen expression was not significantly altered, while levels of connective tissue growth factor (CTGF), IGF-IEa, transforming growth factor-β1, -2 (TGF-β), and the TGF-β receptor II mRNA were increased (P < 0.05). Matrix components tenascin-C, fibronectin, and decorin were also induced in loaded muscle (P < 0.05), while fibromodulin was unaffected. In conclusion, the relatively robust changes in matrix components and related growth factors in muscle indicate a stimulation of extracellular matrix even with moderate exercise. However, in tendon tissue, this exercise model does not appear to induce any anabolic response on the transcriptional level.

Michael Gundill montre comment sauver son dos (part 2)

28/10/2011

 

Mieux comprendre les bénéfices de l’échauffement

27/10/2011

 

Neural conduction and excitability following a simple warm up
Journal of Science and Medicine in Sport   Alan J. Pearce

Objective: This study examined the effect of a generic, active warm up on neural and muscular conduction time. Design: Single group, pre-post design. Method: Central and peripheral neuromuscular conduction time was quantified in the abductor pollicis brevis (APB) and gastrocnemius muscles of 18 healthy participants (mean age 25.9 ± 5.8 years, 12 males) using transcranial magnetic stimulation (TMS) and M-wave techniques, prior to and immediately following an active warm up consisting of 5 min running at 65% of maximum heart rate. Neural conduction time, for both TMS and M-wave, was quantified as the time between stimulus artefact and deflection of the wave form, whilst muscle conduction time for TMS and M-wave, was quantified from the stimulus artefact to the absolute peak twitch response. Results: Following the warm up protocol, a significant reduction in muscle conduction time was found in both TMS and M-wave of 0.43 ms (P = 0.02) and 0.30 ms (P = 0.001) for the APB; and 0.29 ms (P < 0.001) and 0.87 ms (P = 0.003) for the gastrocnemius, respectively. No change was found in neural conduction using either TMS or M-wave techniques. Conclusions: These findings support previous data which demonstrate an improvement in muscular conduction time and subsequent improvement in athletic performance post warm up. The data also make evident that changes in muscular conduction time are a global response to warm up and are not directly related to muscular activity. In contrast, neural conduction time did not change and should not be confused with changes in muscular conduction time in the literature.

Michael Gundill montre comment sauver son dos

25/10/2011

 

Impacts des ultrasons sur l’IGF musculaire

14/10/2011

 

Therapeutic Ultrasound Affects IGF-1 Splice Variant Expression in Human Skeletal Muscle
Diana C. Delgado-Diaz
Am J Sports Med October 2011 vol. 39 no. 10 2233-2241


Background: Animal models of skeletal muscle damage and repair demonstrate that therapeutic ultrasound (TUS) enhances muscle force recovery after damage, increases satellite cell proliferation, and decreases insulin-like growth factor (IGF)-1 splice variant (mechano growth factor) gene expression. However, these effects have not been verified in humans.

Purpose: This study was undertaken to examine the 3 known splice variants of the IGF-1 gene in human skeletal muscle after damage and TUS treatment.

Study Design: Controlled laboratory study.

Methods: Sixteen healthy men (18-29 years of age), physically active, were randomized to either a control (CON) or experimental group (EXP). The EXP group underwent 200 lengthening contractions (muscle damage) of the quadriceps of both legs, 48 hours before TUS. Both groups received TUS, delivered for 10 minutes on a standardized area of the vastus lateralis of only 1 leg (1.0 MHz, 1.5 W/cm2). Bilateral muscle biopsy samples were taken from all participants, 6 hours after TUS. Total RNA was extracted, and quantitative real-time polymerase chain reaction conducted for each IGF-1 splice variant.

Results: Muscle damage was confirmed by a decrease in the isometric peak torque and increase in creatine kinase activity levels 48 hours after damage (P < .01). After muscle damage, gene expression of total IGF-1 and 2 IGF-1 splice variants increased. Therapeutic ultrasound induced significant increase in IGF-1Eb gene expression in undamaged muscle (1.4 ± 0.2-fold, P < 0.01). In damaged skeletal muscle, no significant change in gene expression attributable to TUS was determined.

Conclusion: Insulin-like growth factor–1 splice variants are differentially regulated in human skeletal muscle in response to exercise-induced muscle damage and TUS treatment. A single treatment of TUS in damaged muscle induces no change in the gene expression of the 3 IGF-1 splice variants in humans. In contrast, in undamaged skeletal muscle, TUS significantly increased IGF-1Eb splice variant gene expression.

Clinical Relevance: These findings suggest that TUS may have additional therapeutic uses beyond its current common practice but may not be effective for muscle injury treatment in a young, healthy population.

La force, c’est pas bon pour le coeur

24/09/2011

 

On s’en serait douté vu le nombre de mecs qui meurent de problèmes cardiaques. Mais pour ceux qui en doutent encore :

STRONGMEN SPORT IS ASSOCIATED WITH LARGER ABSOLUTE HEART SIZE AND IMPAIRED CARDIAC
RELAXATION

TOMAS VENCKUNAS
J Strength Cond Res 25(10): 2919–2925, 2011—
This study
was carried out to compare cardiac structure and function and
blood lipids among Strongmen, sedentary controls, and
marathoners. Echocardiography was performed, and endothelial
function, blood lipids and maximal oxygen uptake were
measured in 27 Caucasian adult men (8 Strongmen, 10
marathoners, 9 controls). Absolute cardiac size parameters
such as left ventricular (LV) diameter and wall thickness of
Strongmen were higher (p , 0.05), but relative (body surface
area indexed) parameters were not different between controls
and Strongmen. In Strongmen, the relative LV diameter (p ,
0.05), wall thickness (p,0.001), and LV mass index (p,0.01)
were lower than in marathoners. The absolute but not relative
right ventricular diameter was larger in Strongmen as compared
with controls, whereas all of the measured relative cardiac size
parameters were higher in marathoners as opposed to in
controls. The endothelial function and the ratio of wall thickness
to chamber diameter were similar among the groups (p .0.05).
Maximal oxygen uptake of Strongmen was lower than in
controls (p , 0.05) and marathoners (p , 0.001). Global
diastolic LV function of Strongmen was impaired in comparison
to controls (p , 0.05) and marathoners (p , 0.05). Plasma
lipids were not different between Strongmen and sedentary
controls, but in comparison to runners, Strongmen had higher
low-density lipoprotein-cholesterol (p , 0.05) and lower highdensity
lipoprotein cholesterol (p , 0.01). Participation in
Strongmen sport is associated with higher absolute but not
relative cardiac size parameters, impaired myocardial relaxation,
and low cardiorespiratory fitness. Therefore, Strongmen may
demand greater attention as an extreme group of athletes with
regard to cardiovascular risk.

INTRODUCTION
During endurance running, the heart has to adapt
to both increased volume and pressure load (20).
Long distance runners tend to have an increase
in both the left ventricular (LV) diameter and LV
wall thickness, that is, develop eccentric hypertrophy (11),
also called a symmetric remodeling of the myocardium (6). In
contrast to pathological forms, cardiac hypertrophy because
of endurance training is not associated with impaired LV
function (22) and even a positive relationship between LV
hypertrophy and diastolic function has been reported in
endurance-trained individuals (16).
During heavy strength and power training, hemodynamic
loading conditions differ from those induced by endurance
training and are characterized by marked overshoots of
arterial blood pressure (18). Strength and power training
likely has little or no effect on the size LV diameter (2),
although evidence of strength training effects on cardiac wall
hypertrophy is less consistent. Some studies report unchanged
LV wall thickness (21) and others present thickened
LV wall (2,4).
The evidence on LV function of strength/power athletes is
also contradictory with some (2) but not all studies (4,25)
reporting deteriorated diastolic function. Most of the evidence
on the cardiac parameters of strength/power athletes has been
obtained from bodybuilders or power lifters (2,4).
Strongmen sport is quite a unique and popular non-
Olympic ‘‘pure strength’’ sport where athletes are not
restricted to body weight categories and implied to have
the greatest absolute strength among humans; also, because
they are not strictly persecuted by WADA, the anabolic
steroid or other banned substance usage among them is
believed to be widespread and intense. In addition, anecdotal
evidence about the avoidance of the aerobic exercise and
copious amounts of usually not very healthy foods consumed
may lead one to think of the lifestyle of Strongmen as not
really healthy and thus warrants to consider them as a group
of athletic individuals with increased risk for cardiovascular
disease and other health problems.
Also, Strongmen with their Herculean strength conduct
extremely arduous exercises such as lifting, holding, carrying
(hundreds of kilograms), pulling, and pushing (tons) during
training and competitions, and such tasks trigger a pronounced
cardiac pressure overload and, when conducted
over an extended period of time, might be expected to impact
cardiac size and function to a greater extent than participation
in other types of resistance sport.

As Strongmen’s cardiovascular fitness and health are not
better and some aspects of it seem to be deteriorated as
compared not only to endurance runners but also to
sedentary, athletes who are seriously engaged in Strongman
sport may demand greater attention as an extreme group of
athletic individuals with regard to cardiovascular disease risk.
The recommendations may include cardiovascular disease
risk follow-up on a regular basis, a modification of lifestyle
such as changes in diet and its supplementation consumed,
and inclusion in the daily regimen at least small amounts
of endurance training, especially during off-season or when
the sport career is over.

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